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内蒙古西沙德盖钼矿床锆石U-Pb和辉钼矿Re-Os年龄及其地质意义
内蒙古西沙德盖钼矿是华北地台北缘成矿带上的一处中型斑岩钼矿床.采用锆石LA-ICP-MS铀-铅同位素测年技术,针对其含矿钾长花岗岩和矿石进行的成岩成矿年代学测定.获得西沙德盖似斑状钾长花岗岩体的成岩年龄为245 Ma±10 Ma;通过对7件辉钼矿样品进行Re-Os同位素分析,获得同位素模式年龄介于223.2 Ma～232.6 Ma,等时线年龄为225.4 Ma±2.6 Ma(MSWD=0.30);两种方法获得的年龄相近(成矿年龄略小于成岩年龄),成岩与成矿作用均发生于中三叠世末,属印支期碰撞后伸展环境下构造?岩浆活动的产物.结合对已有年龄资料的归纳和分析,认为区内在印支期曾发生过重要的岩浆-(金钼)成矿事件,该事件虽可能遭受了燕山期构造-岩浆活动的改造或破坏,但在一定区域和一定程度上仍有记录或保留.
ZHANG Yong-mei
GU Xue-xiang
DONG Shu-yi
CHENG Wen-bin
HUANG Zhi-quan
LI Fu-liang
YANG Wei-long
作者单位：
中国地质大学(北京)地质过程与矿产资源国家重点实验室,北京,100083
中国地质大学(北京)地质过程与矿产资源国家重点实验室,北京 100083;成都理工大学地球科学学院,四川成都610059
中国人民武装警察部队黄金第二支队,内蒙古呼和浩特,010010
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国家自然科学基金，国家重点基础研究发展计划“973”项目，长江学者和创新团队计划，高等学校学科创新引智计划，中国人民武装警察部队科研项目，中央高校基本科研业务费专项资金资助
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Personal informationName : Zhi-hui FengDay of birth : Dec, 1983E-mail: &&&&&&&&&&& zhfeng@mail. &&Employment&2012 C PresentCenter for Mitochondrial Biology and Medicine, Frontier Institute of Science and Technology (FIST), Xi’an Jiaotong University, China&2009 C PresentDirector’s Assistant, Institute of Mitochondrial Biology and Medicine, Xi’an Jiaotong University, China
Education &&& Ph.D Insititute for nutritional sciences, Shanghai insitutes for biological Sciences, Chinese Academy of Sciences, Shanghai, ChinaSuperviser : Dr. Yan C Dr. Jiankang Liu&&&& Visiting student Institute of Mitochondrial Miology and Medicine, Xi’an Jiaotong University, Xi’an, China2009&&&&&&&&&&&&&& Visiting student Department of Urology, Chao Family Cancer Center, UC Irvine Medicial Center, USA&2001 - 2005&&& BS Marine College, Shandong University, Weihai, China.&
&Activities2011 Organizing committee, International Symposium of Mitochondrial Biology and Medicine & Chinese-Mit’2011. 2011 Organizing committee, Nestlé Research Center-Xi’an Jiaotong University Nutrition and Health Laboratory Opening Ceremony. 2008 - 2009 Establishment of Institute of Mitochondrial biology and Medicine (IMBM), Xi’an, China
Honors2011 Outstanding student Nestlé prize, Nestlé Research Center and Xi’an Jiaotong University Joint Laboratory2011 Oxygen Club of California Young Investigator Prize, International Symposium on Mitochondrial Biology and Medicine2010 Excellent student awards, Institute of mitochondrial biology and medicine, Xi’an Jiaotong University2004 Shandong University excellent student awards2003 Shandong University excellent student awards2001 Shandong University excellent student awards&
Research experience1. Mitochondrial targeting of nutrtional intervention on age related macular degeneration (AMD). Degeneration of retinal pigment epithelium (RPE) cell is considered major cause that induces AMD, while oxidative stress induced mitochondrial dysfunction is closely related RPE degeneration. Using ARPE-19 cell model, I have selected several nutritients that prevent oxidative stress induced RPE cell death by activating mitochondrial biogenesis, improving mitochondrial function and inducing phase II enzymes.&2. Mitochondrial dynamic regulation in obesity, diabetic development and effects of nutrional intervention. Mitochondrial dysfunction is associated with development of obesity and diabetes. I’m interested in finding out how mitochondrial dynamic including mitochondrial biogenesis, degradation, fusion, fission affect mitochondrial function during obesity and diabetic progression. Using several cell and animal models, I intent to find effective nutritients which could inhibit obesity, diabetes, and associated fatty liver development by regulating mitochondrial function.&3. Involvement of mitochondrial dysfunction in prenatal stress induced offspring congivitive decline and effects of nutritional intervention. It has been well indicated that prenatal stress could induce offspring cognitive decline. My study showed that prenatal stress induced mitochondrial dysfunction and oxidative damage in offspring hippocampus might be the major cause. Besides the well-know role in children brain development, I found novel role of docosahexaenoic acid (22 :6n-3 ; DHA) in protecting prenatal stress induced cognitive dysfuntion involving the modulation of mitochondrial functions and dynamics.&4. Autophagy and mitochondrial remodeling in long-term strenuous exercise. Physical exercise is considered to exert a positive effect on health. While, I found strenuous or excessive exercise causes fatigue and damage to muscle and immune functions thereby decrease endurance capacity. I’m interested in cross talking between autophay and mitochonrial dynamic during exercise.Research Profile (&or )&
Representive Publications (*corresponding author)
Research ID:& E-
website link:&
1. Zheng A, Li H, Cao K, Xu J, Zou X, Li Y, Chen C, Liu J*, Feng Z*. Maternal hydroxytyrosol administration improves neurogenesis and cognitive function in prenatally stressed offspring. J Nutr Biochem. 2014 Nov 12. pii: S)00224-1. doi: 10.1016/j.jnutbio..
2. Cao K, Zheng A, Xu J, Li H, Liu J, Peng Y, Long J, Zou X, Li Y, Chen C, Liu J*, Feng Z*. AMPK activation prevents prenatal stress-induced cognitive impairment: modulation of mitochondrial content and oxidative stress. Free Radic Biol Med. -66.
3. Chen C, Han X, Zou X, Li Y, Yang L, Cao K, Xu J, Long J, Liu J*, Feng Z*.
4-methylene-2-octyl-5-oxotetrahydrofuran-3-carboxylic acid (C75), an inhibitor of fatty-acid synthase, suppresses the mitochondrial fatty acid synthesis pathway and impairs mitochondrial function. J Biol Chem. 2014 Jun 13;289(24):17184-94.
4. Xu J, Cao K, Li Y, Zou X, Chen C, Szeto IM, Dong Z, Zhao Y, Shi Y, Wang J, Liu J*, Feng Z*. Bitter gourd inhibits the development of obesity-associated fatty liver in C57BL/6 mice fed a high-fat diet. J Nutr. ):475-83.
5. Zou X, Yan C, Shi Y, Cao K, Xu J, Wang X, Chen C, Luo C, Li Y, Gao J, Pang W, Zhao J, Zhao F, Li H, Zheng A, Sun W, Long J, Szeto IM, Zhao Y, Dong Z, Zhang P, Wang J, Lu W, Zhang Y, Liu J*, Feng Z*. Mitochondrial dysfunction in obesity-associated nonalcoholic fatty liver disease: the protective effects of pomegranate with its active component punicalagin. Antioxid Redox Signal. 2014 Oct 10;21(11):1557-70.
6. Cao K, Xu J, Zou X, Li Y, Chen C, Zheng A, Li H, Li H, Szeto IM, Shi Y, Long J, Liu J*, Feng Z*.Hydroxytyrosol prevents diet-induced metabolic syndrome and attenuates mitochondrial abnormalities in obese mice. Free Radic Biol Med. -407.
7. Feng Z, Zou X, Jia H, Li X, Zhu Z, Liu X, Bucheli P, Ballevre O, Hou Y, Zhang W, Wang J, Chen Y, Liu J. Maternal docosahexaenoic acid feeding protects against impairment of learning and memory and oxidative stress in prenatally stressed rats: possible role of neuronal mitochondria metabolism. Antioxid Redox Signal. 2012 Feb 1;16(3):275-89.
8. Feng Z, Bai L, Yan J, Li Y, Shen W, Wang Y, Wertz K, Weber P, Zhang Y, Chen Y, Liu J. Mitochondrial dynamic remodeling in strenuous exercise-induced muscle and mitochondrial dysfunction: regulatory effects of hydroxytyrosol. Free Radic Biol Med. 2011 May 15;50(10):1437-46.
9. Feng Z, Liu Z, Li X, Jia H, Sun L, Tian C, Jia L, Liu J. α-Tocopherol is an effective Phase II enzyme inducer: protective effects on acrolein-induced oxidative stress and mitochondrial dysfunction in human retinal pigment epithelial cells. J Nutr Biochem. ):1222-31.
招收硕士生、博士生和博士后。
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